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148498-78-6

中文名稱 TYR-ARG-GLN-SER-MET-ASN-ASN-PHE-GLN-GLY-LEU-ARG-SER-PHE-GLY-CYS-ARG-PHE-GLY-THR-CYS-THR-VAL-GLN-LYS-LEU-ALA-HIS-GLN-ILE-TYR-GLN-PHE-THR-ASP-LYS-ASP-LYS-ASP-ASN-VAL-ALA-PRO-ARG-SER-LYS-ILE-SER-PRO-GLN-GLY-TYR-NH2(DISULFIDE BRIDGE:CYS16-CYS21)
英文名稱 ADRENOMEDULLIN (HUMAN)
CAS 148498-78-6
分子式 C264H406N80O77S3
分子量 6028.73
MOL 文件 148498-78-6.mol
更新日期 2024/10/24 11:51:23
148498-78-6 結(jié)構(gòu)式 148498-78-6 結(jié)構(gòu)式

基本信息

中文別名
YY-52-NH2
腎上腺髓質(zhì)素(ADM)(1~12),人
ADRENOMEDULLIN (1-52) (人)
英文別名
ADM 1-52, HUMAN
humanadrenomedullin
ADRENOMEDULLIN (HUMAN)
adrenomedullin 52 human
ADRENOMEDULLIN 1-52, HUMAN
Human adrenomedullin-(1-52)-NH2
Adrenomedullin (human)/ADM (1-52) (human), Adrenomedullin (1-52), human
YRQSMNNFQGLRSFGCRFGTCTVQKLAHQIYQFTDKDKDNVAPRSKISPQGY-NH2 (DISULFIDE BRIDGE: 16-21)
Tyr-Arg-Gln-Ser-Met-Asn-Asn-Phe-Gln-Gly-Leu-Arg-Ser-Phe-Gly-Cys-Arg-Phe-Gly-Thr-Cys-Thr-Val-Gln-Lys-Leu-Ala-His-Gln-Ile-Tyr-Gln-Phe-Thr-Asp-Lys-Asp-Lys-Asp-Asn-Val-Ala-Pro-Arg-Ser-Lys-Ile-Ser-Pro-Gln-
TYR-ARG-GLN-SER-MET-ASN-ASN-PHE-GLN-GLY-LEU-ARG-SER-PHE-GLY-CYS-ARG-PHE-GLY-THR-CYS-THR-VAL-GLN-LYS-LEU-ALA-HIS-GLN-ILE-TYR-GLN-PHE-THR-ASP-LYS-ASP-LYS-ASP-ASN-VAL-ALA-PRO-ARG-SER-LYS-ILE-SER-PRO-GLN-GLY-TYR-NH2

物理化學(xué)性質(zhì)

儲存條件-20°C
溶解度在1%乙酸中溶解度為1mg/ml
形態(tài)powder
序列H-Tyr-Arg-Gln-Ser-Met-Asn-Asn-Phe-Gln-Gly-Leu-Arg-Ser-Phe-Gly-Cys-Arg-Phe-Gly-Thr-Cys-Thr-Val-Gln-Lys-Leu-Ala-His-Gln-Ile-Tyr-Gln-Phe-Thr-Asp-Lys-Asp-Lys-Asp-Asn-Val-Ala-Pro-Arg-Ser-Lys-Ile-Ser-Pro-Gln-Gly-Tyr-NH2(Disulfide bond)

安全數(shù)據(jù)

危險(xiǎn)性符號(GHS)GHS hazard pictograms
GHS08
警示詞警告
危險(xiǎn)性描述H361
WGK Germany3
RTECS號AV6800000
海關(guān)編碼2937190000

常見問題列表

生物活性
Adrenomedullin (AM) (1-52), human 是一種含有 52 個(gè)氨基酸的多肽,影響癌細(xì)胞增殖和血管生成。
體外研究

To explore the effect of Adrenomedullin (AM) (1-52), human on astroglioma cells, CRT-MG cells are incubated in the absence or presence of Adrenomedullin (AM) (1-52), human (ADM1-52) for 48 h in a medium containing 1% FBS, and wound-healing assay is performed. The number of cells migrating to the wound region significantly increases in the ADM1-52-treated cells, in a dose-dependent manner, compared to the untreated cells.

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