| Identification | Back Directory | [Name]
MAF | [CAS]
101365-54-2 | [Synonyms]
MAF
C-Maf
4hopcp
4-ho-pcp
MAF power
IFN-γ human
Immune Interferon
T cell interferon
type II interferon
Proto-oncogene c-maf
Methoxyacetyl fentanyl
fuef CAS NO.101365-54-2
Ferric methylarsonate
Transcription factor Maf
4CLMPD CAS NO.101365-54-2
maf Methoxyacetylfentanyl
methoxyaceylfentanyl MAF
2-METHYL-METHOXYACETYLFENTANYL
MAF 5F-MDMB-2201 methoxyaceylfentanyl
Methoxyacetyl fentanyl (hydrochloride)
5F-MDMB-2201 4CLMPD CAS NO.101365-54-2
good quality 99.5% MAF with lowest price
Methoxyacetyl fentanyl (hydrochloride) (CRM)
Methoxyacetyl fentanyl hydrochloride solution
Anti-MAF, C-Terminal antibody produced in rabbit
V-maf musculoaponeurotic fibrosarcoma oncogene homolog | [Molecular Formula]
C3H9As3Fe2O9 | [MOL File]
101365-54-2.mol | [Molecular Weight]
525.553 |
| Hazard Information | Back Directory | [Uses]
IFN-γ (interferon γ)-human has been used for the generation of inflammatory M1 cells from human monocytic cell line, THP1. | [General Description]
IFN-γ (interferon γ) is an acid-labile glycosylated interferon produced by CD4 and CD8 T lymphocytes as well as activated NK (natural killer) cells. IFN-γ receptors are present in most immune cells, which respond to IFN-γ signaling by increasing the surface expression of class I MHC (major histocompatibility complex) proteins. This promotes the presentation of antigen to T-helper (CD4+) cells. The gene is mapped to human chromosome 12q15. Human IFN-γ is species-specific and is biologically active only in human and primate cells. Recombinant human IFN-γ is a 16.7kDa protein containing 143 amino acid residues. | [Biochem/physiol Actions]
IFN-γ (interferon γ) signaling in antigen-presenting cells and antigen-recognizing B and T lymphocytes regulate the antigen-specific phases of the immune response. It can suppress the generation of anti-inflammatory cytokines and enhance the secretion of proinflammatory cytokines. Additionally, IFN-γ stimulates a number of lymphoid cell functions including the anti-microbial and anti-tumor responses of macrophages, NK (natural killer) cells and neutrophils. It is responsible for tumor rejection and is capable of killing tumor cells by autophagy or apoptosis. IFN-γ is also involved in Hepatitis E virus disease pathogenesis. |
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